IMMUNO-INFLAMMATORY BIOMARKERS OF CARDIORENAL SYNDROME
DOI:
https://doi.org/10.5281/zenodo.11194883Abstract
Renal dysfunction is associated with a higher incidence of recurrence of myocardial ischemia, myocardial infarction (MI), stroke, serious hemorrhagic complications, acute heart failure, atrial and ventricular fibrillation. Even a slight decrease in kidney function significantly worsens the course of the underlying cardiac pathology, at the same time angiotensin converting enzyme, angiotensin II receptor antagonists, nitrates, methods of renal replacement therapy) increasing the incidence of complications and the risk of death, and, conversely, a decrease in myocardial contractile function affects the kidneys in the most negative way. The need for early detection of kidney damage in cardiovascular pathology in order to assess risk, develop strategies and tactics for patient management contributed to the emergence of such concepts as "cardiorenal anemia syndrome" and "cardiorenal continuum" [1,2].
References
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